@article{leak_tove_parks_1999, title={In yeast, upc2-1 confers a decrease in tolerance to LiCl and NaCl, which can be suppressed by the P-type ATPase encoded by ENA2}, volume={18}, ISSN={["1044-5498"]}, DOI={10.1089/104454999315510}, abstractNote={Wild-type yeast cells are unable to take up sterols from their growth media under aerobic conditions and are relatively resistant to monovalent cations. A yeast mutant (upc2-1) with a defect in the aerobic exclusion of sterols was found to have increased sensitivity to LiCl and NaCl. Although cation sensitivity has been reported for mutants that synthesize altered sterols, the mutant with upc2-1 continues to produce the normal sterol, ergosterol. The ENA2 gene was cloned on the basis of remediating the hypersensitivity to the monovalent cations.}, number={2}, journal={DNA AND CELL BIOLOGY}, author={Leak, FW and Tove, S and Parks, LW}, year={1999}, month={Feb}, pages={133–139} }
 @article{crowley_leak_shianna_tove_parks_1998, title={A mutation in a purported regulatory gene affects control of sterol uptake in Saccharomyces cerevisiae}, volume={180}, number={16}, journal={Journal of Bacteriology}, author={Crowley, J. H. and Leak, F. W. and Shianna, K. V. and Tove, S. and Parks, L. W.}, year={1998}, pages={4177–4183} }
 @article{palermo_leak_tove_parks_1997, title={Assessment of the essentiality of ERG genes late in ergosterol biosynthesis in Saccharomyces cerevisiae}, volume={32}, ISSN={["1432-0983"]}, DOI={10.1007/s002940050252}, abstractNote={Isogenic strains of yeast were constructed, differing only in insertionally inactivated genes for ergosterol biosynthesis. These and their allelic wild-types were grown in competition to ascertain growth differences and any selective advantage for organisms producing sterols with or without specific features of ergosterol. In every instance tested, the wild-type allele afforded a competitive advantage over the isogenic pair producing modified sterol structures instead of ergosterol. A general trend was seen in which the earlier in the biosynthetic pathway that a mutation occurred, the less able the strain producing the defective sterols could compete with the ergosterol-producing strains.}, number={2}, journal={CURRENT GENETICS}, author={Palermo, LM and Leak, FW and Tove, S and Parks, LW}, year={1997}, month={Aug}, pages={93–99} }