Yuehua Li

Protease-activated receptor-2 (PAR-2) is a weak enhancer of mucin secretion by human bronchial epithelial cells in vitro

[Review of ]. INTERNATIONAL JOURNAL OF BIOCHEMISTRY & CELL BIOLOGY, 40(6-7), 1379–1388.

Method and compositions for altering mucus secretion

Washington, DC: U.S. Patent and Trademark Office.

(R)-Albuterol elicits antiinflammatory effects in human airway epithelial cells via iNOS

American Journal of Respiratory Cell and Molecular Biology, 34(1), 119–127.

Methods for regulating inflammatory mediators and peptides useful therein

Washington, DC: U.S. Patent and Trademark Office.

Human neutrophil elastase induces hypersecretion of mucin from well-differentiated human bronchial epithelial cells in vitro via a protein kinase C delta-mediated mechanism

AMERICAN JOURNAL OF PATHOLOGY, 167(3), 651–661.

A MARCKS-related peptide blocks mucus hypersecretion in a mouse model of asthma

NATURE MEDICINE, 10(2), 193–196.

Blocking peptide for inflammatory cell secretion

Washington, DC: U.S. Patent and Trademark Office.

A myristoylated peptide directed against the N-terminal region of MARCKS protein inhibits mucin secretion in ovalbumin sensitized/challenged mice in vivo.

American Journal of Respiratory and Critical Care Medicine, Vol. 167, p. A17.

Activation of protease-activated receptors?2 (PAR-2) is not associated with enhanced mucin secretion by well-differentiated normal human bronchial epithelial cells in vitro.

American Journal of Respiratory and Critical Care Medicine, Vol. 167, p. A204.

Differential effects of albuterol isomers on normal human bronchial epithelial cells in vitro.

American Journal of Respiratory and Critical Care Medicine, Vol. 167, p. A205.

Human neutrophil elastase provokes release of MUC5B mucin from normal bronchial epithelial cells in vitro via a PKC-dependent mechanism.

American Journal of Respiratory and Critical Care Medicine, Vol. 167, p. A203.

Airway epithelium and mucus - Intracellular signaling pathways for gene expression and secretion

AMERICAN JOURNAL OF RESPIRATORY CELL AND MOLECULAR BIOLOGY, 25(4), 397–400.

Enhanced expression of mucin genes in a guinea pig model of allergic asthma

AMERICAN JOURNAL OF RESPIRATORY CELL AND MOLECULAR BIOLOGY, 25(5), 644–651.

MARCKS protein interaction with the cellular contractile machinery may regulate mucin secretion by human airway epithelium.

American Journal of Respiratory and Critical Care Medicine, Vol. 163, p. A225.

MARCKS protein is a key molecule regulating mucin secretion by human airway epithelial cells in vitro

JOURNAL OF BIOLOGICAL CHEMISTRY, 276(44), 40982–40990.

MARCKS protein: a key intracellular molecule controlling mucin secretion by human airway goblet cells.

American Journal of Respiratory and Critical Care Medicine, Vol. 161, p. A259.

Myristoylated alanine-rich C-kinase substrate protein: A major intracellular regulatory molecule controlling secretion of mucin by human airway goblet cells.

Chest, Vol. 117, pp. 266S–267S.

Residual oil fly ash induces cytotoxicity and mucin secretion by guinea pig tracheal epithelial cells via an oxidant-mediated mechanism

TOXICOLOGY AND APPLIED PHARMACOLOGY, 163(3), 221–230.

Myristoylated alanine-rich C kinase substrate (MARCKS) is produced by human airway epithelial cells and is phosphorylated by PKC and PKG.

American Journal of Respiratory and Critical Care Medicine, Vol. 159, p. A723.

Residual oil fly ash (ROFA) increases mucin secretion and mucin gene expression in guinea pig airway epithelial cells in vitro.

American Journal of Respiratory and Critical Care Medicine, Vol. 159, p. A888.

TNF?-induced ICAM-1 expression in airway epithelium: involvement of IkB?.

American Journal of Respiratory and Critical Care Medicine, Vol. 159, p. A184.

Cloning of guinea pig Muc2 cDNA and MUC2 gene expression in guinea pig airway epithelium in vitro.

American Journal of Respiratory and Critical Care Medicine, Vol. 157, p. A728.