Feng Zhu

Works (3)

Updated: July 5th, 2023 21:15

2011 journal article

CD34 antigen: Determination of specific sites of phosphorylation in vitro and in vivo

INTERNATIONAL JOURNAL OF MASS SPECTROMETRY, 301(1-3), 12–21.

By: L. Deterding *, J. Williams *, M. Humble*, R. Petrovich *, S. Wei *, C. Trempus*, M. Gates*, F. Zhu n ...

author keywords: CD34; Phosphorylation; PKC kinase; AKT2 kinase; LC/MS/MS
TL;DR: The identification of the in vitro sites of phosphorylation on the intracellular domain of mouse CD34 following PKC treatment suggests that other kinases, as well as PKC, may have important signaling functions in CD34. (via Semantic Scholar)
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Sources: Web Of Science, NC State University Libraries
Added: August 6, 2018

2009 journal article

Subcellular localization of APMCF1 and its biological significance of expression pattern in normal and malignant human tissues

Journal of Experimental & Clinical Cancer Research, 28.

By: Y. Zhang, Q. Li, F. Zhu, J. Cui, K. Li, Q. Li, R. Wang, W. Wang, W. Wang, W. Yan

Source: NC State University Libraries
Added: August 6, 2018

2008 journal article

C/EBP beta represses p53 to promote cell survival downstream of DNA damage independent of oncogenic Ras and p19(Arf)

CELL DEATH AND DIFFERENTIATION, 15(11), 1734–1744.

By: S. Ewing n, S. Zhu n, F. Zhu n, J. House n  & R. Smart n 

Contributors: S. Ewing n, S. Zhu n, F. Zhu n, J. House n  & R. Smart n 

author keywords: apoptosis; p53; C/EBP beta; p19(Arf); DNA damage; keratinocytes
MeSH headings : Animals; Apoptosis / drug effects; CCAAT-Enhancer-Binding Protein-beta / deficiency; CCAAT-Enhancer-Binding Protein-beta / metabolism; Carcinogens / toxicity; Cell Survival / drug effects; Cyclin-Dependent Kinase Inhibitor p16 / metabolism; Cyclophosphamide / administration & dosage; Cyclophosphamide / pharmacology; DNA Damage; Epidermal Cells; Epidermis / drug effects; Epidermis / metabolism; Injections, Intraperitoneal; Keratinocytes / cytology; Keratinocytes / drug effects; Keratinocytes / metabolism; Mice; Oncogene Protein p21(ras) / metabolism; Protein Processing, Post-Translational / drug effects; Repressor Proteins / metabolism; Transcriptional Activation / drug effects; Tumor Suppressor Protein p53 / genetics; Tumor Suppressor Protein p53 / metabolism
TL;DR: Results suggest that inhibition of C/EBPβ may be a target for cancer cotherapy to increase the efficacy of alkylating chemotherapeutic agents and suggest that p19Arf represses p53 to promote cell survival downstream of DNA damage. (via Semantic Scholar)
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Sources: Web Of Science, NC State University Libraries, ORCID
Added: August 6, 2018

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