@article{sergeeff_armstrong_bunch_2004, title={Hepatic abscesses in cats: 14 cases (1985-2002)}, volume={18}, ISSN={["1939-1676"]}, DOI={10.1892/0891-6640(2004)18<295:HAICC>2.0.CO;2}, abstractNote={In this retrospective study, we describe 14 cats diagnosed with hepatic abscesses. The objective of the study was to report the clinical signs, physical examination findings, clinicopathologic findings, and outcomes in affected cats. These findings were then compared with those previously reported in dogs and humans. Clinical signs were vague and included anorexia, lethargy, and weight loss. Only 23% of cats had fever, whereas 31% were hypothermic. Increases in serum activities of alanine aminotransferase and alkaline phosphatase were found in 45 and 18%, respectively, of the 11 cats that had laboratory work performed. Abdominal ultrasound examinations were performed in 7 cats, and abnormalities were found in 71% of them. Four cats had solitary abscesses, all of which were located in the right liver lobes. The other 10 cats had multifocal small abscesses or microabscesses, and all of these cats had clinical signs suggestive of sepsis. Cytologic evaluation of samples obtained by abdominocentesis indicated septic inflammation in 67% of cats in which peritoneal fluid was analyzed. Hepatic abscess cultures yielded polymicrobial growth in 66% of the cats: Escherichia coli was the most commonly cultured organism. Overall mortality rate was 79%. All survivors underwent exploratory laparotomy for partial hepatectomy to resect the abscess followed by medical management. Hepatic abscesses should be considered in cats with signs consistent with sepsis. More routine use of ultrasonography may aid in earlier diagnosis of hepatic abscesses, potentially improving prognosis and outcome.}, number={3}, journal={JOURNAL OF VETERINARY INTERNAL MEDICINE}, author={Sergeeff, JS and Armstrong, PJ and Bunch, SE}, year={2004}, pages={295–300} }
 @article{bunch_ford_hawkins_jackson_vaden_breitschwerdt_2004, title={The Clinician Investigator Program in Companion Animal Internal Medicine at North Carolina State University}, volume={31}, ISSN={0748-321X 1943-7218}, url={http://dx.doi.org/10.3138/jvme.31.4.425}, DOI={10.3138/jvme.31.4.425}, abstractNote={ A retrospective study was conducted to describe the development and evolution of the combined internal medicine/PhD program, the Clinician Investigator (CI) Program, at North Carolina State University. Separate survey instruments were developed for individuals who had committed to completing both the residency and PhD components and for graduate advisors of individuals who were granted the PhD degree. Results are summarized here. Most CIs reported believing that each component of the program (clinical training and research training) provided mutual benefits and that their teaching skills were enhanced, particularly as a result of instructing students in the Veterinary Teaching Hospital. Opinions among both the CIs and the graduate advisors were divided about the benefits of a combined program compared with a sequential program; however, all but one of 11 CIs who completed the survey would enroll in the combined program again. The graduate advisors were overwhelmingly positive about the CIs they had advised and indicated that they would welcome a CI as a PhD student in their laboratory again. Suggested areas for improvement included guaranteed salary/stipend support for the average time to completion (six years) instead of for five years, as well as more emphasis on and guidance in identifying a graduate advisor earlier in the CI program so as to smooth the transition between the clinical and research training components of the program. It is hoped that other training programs will benefit from the summary of our experience with this program. }, number={4}, journal={Journal of Veterinary Medical Education}, publisher={University of Toronto Press Inc. (UTPress)}, author={Bunch, Susan E. and Ford, Richard B. and Hawkins, Eleanor C. and Jackson, Mark W. and Vaden, Shelly L. and Breitschwerdt, Edward B.}, year={2004}, month={Dec}, pages={425–434} }
 @article{gorlinger_rothuizen_bunch_van den ingh_2003, title={Congenital dilatation of the bile ducts (Caroli's disease) in young dogs}, volume={17}, DOI={10.1892/0891-6640(2003)017<0028:CDOTBD>2.3.CO;2}, abstractNote={We describe 8 young dogs with congenital dilatation of the intra- and extrahepatic bile ducts and diffuse cystic kidney disease, compatible with Caroli's disease in humans. The dogs were referred between 1980 and 2000 because of chronic disease at an age of 6 months to 3 years. These dogs included 3 Collies, 2 Frisian Stabyhouns, 2 Jack Russell Terriers, and 1 mixed-breed dog. The most common signs were vomiting (6/6), polyuria and polydipsia (4/6), and anorexia (4/6). Ascites was a common finding (4/6). Clinicopathologic abnormalities were available for 6 dogs. All had increased plasma alkaline phosphatase activity and fasting bile acids: increased alanine aminotransferase activity and urea and creatinine concentrations were present in 50% of dogs. Ultrasound examination of the liver showed severely dilated bile ducts without evidence of obstruction, and calcification in all cases but 1. Postmortem examination revealed severe dilatation of the larger intra- and extrahepatic bile ducts. The common bile duct and gall bladder were normal, and the bile system was patent. The ducts contained a clear viscid fluid often with calcified material. Microscopically, marked portal fibrosis was present, often with abnormally structured dilated bile ducts lined with columnar or cuboid epithelium and regularly small calcifications. The lesion was complicated by ascending cholangitis in 1 dog. The kidneys showed marked cortical and medullary fibrosis with a diffuse radial cystic pattern; only slight renal fibrosis was found in the oldest dog. Seven dogs were euthanized without treatment; the oldest dog was alive and well 5 months after diagnosis and was maintained on a protein-restricted diet.}, number={1}, journal={Journal of Veterinary Internal Medicine}, author={Gorlinger, S. and Rothuizen, J. and Bunch, S. and Van Den Ingh, T.}, year={2003}, pages={28–32} }
 @article{savary-bataille_bunch_spaulding_jackson_mac law_stebbins_2003, title={Percutaneous ultrasound-guided cholecystocentesis in healthy cats}, volume={17}, ISSN={["1939-1676"]}, DOI={10.1892/0891-6640(2003)017<0298:PUCIHC>2.3.CO;2}, number={3}, journal={JOURNAL OF VETERINARY INTERNAL MEDICINE}, author={Savary-Bataille, KCM and Bunch, SE and Spaulding, KA and Jackson, MW and Mac Law, J and Stebbins, ME}, year={2003}, pages={298–303} }
 @article{seguin_bunch_2001, title={Iatrogenic copper deficiency associated with long-term copper chelation for treatment of copper storage disease in a Bedlington Terrier}, volume={218}, DOI={10.2460/javma.2001.218.1593}, abstractNote={A 9-year-old Bedlington Terrier was evaluated because of weight loss, inappetence, and hematemesis. Copper storage disease had been diagnosed previously on the basis of high hepatic copper concentration. Treatment had included dietary copper restriction and administration of trientine for chelation of copper. A CBC revealed microcytic hypochromic anemia. High serum activities of liver enzymes, high bile acid concentrations, and low BUN and albumin concentrations were detected. Vomiting resolved temporarily with treatment, but the clinicopathologic abnormalities persisted. Results of transcolonic portal scintigraphy suggested an abnormal shunt fraction. Results of liver biopsy and copper quantification revealed glycogen accumulation and extremely low hepatic copper concentration. Serum and hair copper concentrations were also low. Chelation and dietary copper restriction were tapered and discontinued. Clinical signs and all clinicopathologic abnormalities improved during a period of several months.}, number={10}, journal={JOURNAL OF THE AMERICAN VETERINARY MEDICAL ASSOCIATION}, author={Seguin, MA and Bunch, SE}, year={2001}, month={May}, pages={1593-+} }
 @article{bunch_johnson_cullen_2001, title={Idiopathic noncirrhotic portal hypertension in dogs: 33 cases (1982-1998)}, volume={218}, ISSN={["0003-1488"]}, DOI={10.2460/javma.2001.218.392}, abstractNote={Abstract}, number={3}, journal={JOURNAL OF THE AMERICAN VETERINARY MEDICAL ASSOCIATION}, author={Bunch, SE and Johnson, SE and Cullen, JM}, year={2001}, month={Feb}, pages={392–399} }
 @article{cowan_bunch_2001, title={Oral antidiabetic drugs for cats}, volume={23}, number={7}, journal={Compendium on Continuing Education for the Practicing Veterinarian}, author={Cowan, S. M. and Bunch, S. E.}, year={2001}, pages={633–642} }
 @inbook{bunch_2000, title={Acute hepatic disorders and systemic disorders that involve the liver}, ISBN={0721672566}, booktitle={Textbook of veterinary internal medicine : diseases of the dog and cat (5th Ed.)}, publisher={Philadelphia, PA : W.B. Saunders}, author={Bunch, S. E.}, year={2000}, pages={1326} }
 @inbook{hess_bunch_2000, title={Diagnostic approach to hepatobiliary disease}, volume={13}, booktitle={Kirk's current meterinary therapy XIII, small animal practice}, publisher={Philadelphia: WB Saunders Co.}, author={Hess, P. R. and Bunch, S. E.}, year={2000}, pages={659–664} }
 @article{betton_healy_english_bunch_1999, title={Atypical limbal melanoma in a cat}, volume={13}, ISSN={["0891-6640"]}, DOI={10.1892/0891-6640(1999)013<0379:ALMIAC>2.3.CO;2}, number={4}, journal={JOURNAL OF VETERINARY INTERNAL MEDICINE}, author={Betton, A and Healy, LN and English, RV and Bunch, SE}, year={1999}, pages={379–381} }
 @article{gookin_trepanier_bunch_1999, title={Clinical hypothyroidism associated with trimethoprim- sulfadiazine administration in a dog}, volume={214}, number={7}, journal={Journal of the American Veterinary Medical Association}, author={Gookin, J. L. and Trepanier, L. A. and Bunch, S. E.}, year={1999}, pages={1028–1031} }
 @article{gookin_bunch_rush_grindem_1998, title={Evaluation of microcytosis in 18 Shibas}, volume={212}, number={8}, journal={Journal of the American Veterinary Medical Association}, author={Gookin, J. L. and Bunch, S. E. and Rush, L. J. and Grindem, C. B.}, year={1998}, pages={1258–1259} }
 @article{gookin_brooks_catalfamo_bunch_munana_1997, title={Factor X deficiency in a cat}, volume={211}, number={5}, journal={Journal of the American Veterinary Medical Association}, author={Gookin, J. L. and Brooks, M. B. and Catalfamo, J. L. and Bunch, S. E. and Munana, K. R.}, year={1997}, pages={576–579} }
 @article{bunch_jordan_sellon_cullen_smith_1995, title={Characterization of iron status in young dogs with portosystemic shunt}, volume={56}, number={7}, journal={American Journal of Veterinary Research}, author={Bunch, S. E. and Jordan, H. L. and Sellon, R. K. and Cullen, J. M. and Smith, J. E.}, year={1995}, pages={853} }
 @article{cook_breitschwerdt_levine_bunch_linn_1993, title={Risk factors associated with acute pancreatitis in dogs: 101 cases (1985-1990)}, volume={203}, number={5}, journal={Journal of the American Veterinary Medical Association}, author={Cook, A. K. and Breitschwerdt, E. B. and Levine, J. F. and Bunch, S. E. and Linn, L. O.}, year={1993}, pages={673–679} }
 @article{bunch_1991, title={Hepatic encephalopathy}, volume={2}, number={4}, journal={Progress in Veterinary Neurology}, author={Bunch, S. E.}, year={1991}, pages={287} }
 @article{bunch_easley_cullen_1990, title={Hematologic values and plasma and tissue folate concentrations in dogs given phenytoin on a long-term basis}, volume={51}, number={11}, journal={American Journal of Veterinary Research}, author={Bunch, S. E. and Easley, J. R. and Cullen, J. M.}, year={1990}, pages={1865} }