@article{rister_amato_nash_mccoy_bereman_mccoy_2021, title={Toxicant exposure during pregnancy increases protective proteins in the dam and a sexually dimorphic response in the fetus}, volume={413}, ISSN={["1096-0333"]}, DOI={10.1016/j.taap.2021.115407}, abstractNote={Endocrine disrupting compounds (EDCs) are ubiquitous environmental pollutants that alter endocrine system function, induce birth defects, and a myriad of other negative health outcomes. Although the mechanism of toxicity of many EDCs have been studied in detail, little work has focused on understanding the mechanisms through which pregnant dams and fetuses protect themselves from EDCs, or if those protective mechanisms are sexually dimorphic in fetuses. In this study, we examined proteomic alterations in the livers of mouse dams and their male and female fetuses induced by vinclozolin, a model antiandrogenic EDC. Dam livers upregulated nine phase I and phase II detoxification pathways and pathway analysis revealed that more pathways are significantly enriched in dam livers than in fetal livers. Phase I and II detoxification proteins are also involved in steroid and steroid hormone biosynthesis and vinclozolin likely alters steroid levels in both the dam and the fetus. The response of the fetal liver proteome to vinclozolin exposure is sexually dimorphic. Female fetal livers upregulated proteins in xenobiotic metabolism pathways, whereas male fetal livers upregulated proteins in oxidative phosphorylation pathways. These results suggest that female fetuses increase protective mechanisms, whereas male fetuses increase ATP production and several disease pathways that are indicative of oxidative damage. Females fetuses upregulate proteins and protective pathways that were similar to the dams whereas males did not. If this sexually dimorphic pattern is typical, then males might generally be more sensitive to EDCs.}, journal={TOXICOLOGY AND APPLIED PHARMACOLOGY}, author={Rister, Alana L. and Amato, Ciro M. and Nash, Tara and McCoy, Michael W. and Bereman, Michael and McCoy, Krista A.}, year={2021}, month={Feb} }
 @article{beri_nash_martin_bereman_2017, title={Exposure to BMAA mirrors molecular processes linked to neurodegenerative disease}, volume={17}, number={17-18}, journal={Proteomics}, author={Beri, J. and Nash, T. and Martin, R. M. and Bereman, M. S.}, year={2017} }
 @article{reyes_nash_dallas_ascencio-ibanez_hanley-bowdoin_2013, title={Peptide Aptamers That Bind to Geminivirus Replication Proteins Confer a Resistance Phenotype to Tomato Yellow Leaf Curl Virus and Tomato Mottle Virus Infection in Tomato}, volume={87}, ISSN={["1098-5514"]}, DOI={10.1128/jvi.01095-13}, abstractNote={ABSTRACT}, number={17}, journal={JOURNAL OF VIROLOGY}, author={Reyes, Maria Ines and Nash, Tara E. and Dallas, Mary M. and Ascencio-Ibanez, J. Trinidad and Hanley-Bowdoin, Linda}, year={2013}, month={Sep}, pages={9691–9706} }
 @article{fabregas_li_boeren_nash_goshe_clouse_vries_cano-delgado_2013, title={The BRASSINOSTEROID INSENSITIVE1-LIKE3 Signalosome Complex Regulates Arabidopsis Root Development}, volume={25}, ISSN={["1532-298X"]}, DOI={10.1105/tpc.113.114462}, abstractNote={Abstract}, number={9}, journal={PLANT CELL}, author={Fabregas, Norma and Li, Na and Boeren, Sjef and Nash, Tara E. and Goshe, Michael B. and Clouse, Steven D. and Vries, Sacco and Cano-Delgado, Ana I.}, year={2013}, month={Sep}, pages={3377–3388} }