@article{nasisse_glover_moore_weigler_1998, title={Detection of feline herpes virus 1 DNA in corneas of cats with eosinophilic keratitis or corneal sequestration}, volume={59}, number={7}, journal={American Journal of Veterinary Research}, author={Nasisse, M. P. and Glover, T. L. and Moore, C. P. and Weigler, B. J.}, year={1998}, pages={856–858} }
 @article{weigler_wiltron_hancock_thigpen_goelz_forsythe_1998, title={Further evaluation of a diagnostic polymerase chain reaction assay for Pasteurella pneumotropica}, volume={48}, number={2}, journal={Laboratory Animal Science}, author={Weigler, B. J. and Wiltron, L. A. and Hancock, S. I. and Thigpen, J. E. and Goelz, M. F. and Forsythe, D. B.}, year={1998}, pages={193–196} }
 @article{weigler_guy_nasisse_hancock_sherry_1997, title={Effect of a live attenuated intranasal vaccine on latency and shedding of feline herpesvirus 1 in domestic cats}, volume={142}, ISSN={["0304-8608"]}, DOI={10.1007/s007050050250}, abstractNote={A prospective study was conducted that evaluated duration of virus shedding through acute and experimentally-induced recurrent disease episodes in 12 cats, and tissue distribution of latent infections, following intranasal vaccination with a temperature sensitive (ts) mutant strain of feline herpesvirus 1 (FHV1). Six of these cats were challenged with a virulent field strain of the agent to assess the extent to which vaccination affected subsequent shedding of virus and the establishment of latent infections. Virus isolation (VI) tests were done in parallel with a polymerase chain reaction (PCR) assay to compare the performance of each diagnostic method. The PCR confirmed that all 12 cats shed virus throughout the periods of vaccination, challenge or mock-challenge, and a cyclophosphamide-dexamethasone stress protocol to reactivate latent infections. Shedding to the tsFHV1 was documented by VI for up to 25 days following vaccination and for up to 15 days following challenge, but not after experimental stress. Overall, FHV1 was present in 144 of 300 (48%) cat-days of testing by PCR compared to 32 of 300 (11%) by VI. The frequency and distribution of latent FHV1 detected in neurologic, ophthalmic, and other tissues by PCR were identical for vaccine-only and vaccine-challenge groups, thereby disproving previous hypotheses that tsFHV1 mutants administered by this route protect against latency.}, number={12}, journal={ARCHIVES OF VIROLOGY}, author={Weigler, BJ and Guy, JS and Nasisse, MP and Hancock, SI and Sherry, B}, year={1997}, pages={2389–2400} }
 @article{effects of valacyclovir in cats infected with feline herpesvivus 1_1997, volume={58}, number={10}, journal={American Journal of Veterinary Research}, year={1997}, pages={1141–1144} }
 @article{weigler_babineau_sherry_nasisse_1997, title={High sensitivity polymerase chain reaction assay for active and latent feline herpesvirus-1 infections in domestic cats}, volume={140}, ISSN={["0042-4900"]}, DOI={10.1136/vr.140.13.335}, abstractNote={A polymerase chain reaction (PCR) assay was developed and used to detect feline herpesvirus‐1 (FHV‐1) in conjunctival and oropharyngeal swabs, and in latently infected tissues (trigeminal ganglia, optic nerves, optic chiasma, olfactory bulbs and corneas) collected from 10 experimentally infected cats. There was good agreement between parallel tests of the swab specimens by PCR and virus isolation assay during the phase of acute, latent and recurrent disease episodes (kappa=0.63, P<0.001). The PCR reliably detected ≤240 copies of FHV‐1 template DNA, significantly improving upon previously published PCR assays for the agent.}, number={13}, journal={VETERINARY RECORD}, author={Weigler, BJ and Babineau, CA and Sherry, B and Nasisse, MP}, year={1997}, month={Mar}, pages={335–338} }
 @article{baneth_weigler_1997, title={Retrospective case-control study of hepatozoonosis in dogs in Israel}, volume={11}, DOI={10.1111/j.1939-1676.1997.tb00482.x}, abstractNote={Signalment, clinical signs, and physical examination and clinicopathologic findings in dogs diagnosed withHepatozoon cam'sparasitemia (n = 100) were compared with those inHepatozoon‐negative dogs (n = 180). A subset (n = 15) ofHepatozoon‐positive dogs with unusually high (> 800H canisgametocytes/μL of whole blood) parasitemia was compared with dogs that had low parasitemia (n = 85) and with Hepatozoon‐negative dogs (n = 180).Hepatozoon‐positive dogs significantly differed from Hepatozoon‐negative dogs in body temperature, total red blood cell count, hemoglobin concentration, hematocrit, and platelet count. Dogs with highH canisparasitemia significantly differed from those with low parasitemia in hemoglobin concentration, hematocrit, and total neutrophil count. Clinical findings from dogs with highH canisparasitemia included emaciation, lethargy, hyperglobulinemia, hypoalbuminemia, and increased serum alkaline phosphatase and creatine kinase activities. Findings at necropsy included hepatitis, pneumonia, and glomerulonephritis associated withH canisschizonts and extensive parasitism of bone marrow, spleen, and lymph nodes. Low hemoglobin concentration, low platelet count, and concurrent parvovirus infection together represented the best predictor variables forHepatozoonpositivity in dogs presenting to the hospital.}, number={6}, journal={Journal of Veterinary Internal Medicine}, author={Baneth, G. and Weigler, B.}, year={1997}, pages={365–370} }
 @article{weigler_thulin_vandewoude_wolfle_1997, title={The supply and demand for laboratory animal veterinarians from 1980-2005}, volume={36}, number={2}, journal={Contemporary Topics in Laboratory Animal Science}, author={Weigler, B. J. and Thulin, J. D. and Vandewoude, S. and Wolfle, T. L.}, year={1997}, pages={39–46} }
 @article{weigler_thigpen_goelz_babineau_forsythe_1996, title={Randomly amplified polymorphic DNA polymerase chain reaction assay for molecular epidemiology investigation of Pasteurella pneumotropica in laboratory rodent colonies}, volume={46}, number={4}, journal={Laboratory Animal Science}, author={Weigler, B. J. and Thigpen, J. E. and Goelz, M. F. and Babineau, C. A. and Forsythe, D. B.}, year={1996}, pages={386} }
 @article{weigler_ksiazek_vandenbergh_levin_sullivan_1996, title={Serological evidence for zoonotic hantaviruses in North Carolina rodents}, volume={32}, ISSN={["0090-3558"]}, DOI={10.7589/0090-3558-32.2.354}, abstractNote={In a survey of seven species of wild rodents (n = 423) collected between October 1993 and March 1994 from the three principal ecological biomes of North Carolina (USA), we found hantavirus antibodies in seven (2%) of 301 Peromyscus spp. Hantavirus antibodies were detected in P. leucopus and P. maniculatus captured from mountain and coastal island bi-omes. Three mice were positive for Sin Nom-bre virus, while four others had antibodies to Seoul virus or a related agent. Two mice serologically positive for Sin Nombre virus were collected from inside a private mountain domicile. We conclude that the risk of human exposure to hantaviruses in North Carolina resembles that for most other areas of the continental United States.}, number={2}, journal={JOURNAL OF WILDLIFE DISEASES}, author={Weigler, BJ and Ksiazek, TG and Vandenbergh, JG and Levin, M and Sullivan, WT}, year={1996}, month={Apr}, pages={354–357} }
 @article{weigler_scinicariello_hilliard_1995, title={RISK OF VENEREAL-B VIRUS (CERCOPITHECINE HERPESVIRUS-1) TRANSMISSION IN RHESUS-MONKEYS USING MOLECULAR EPIDEMIOLOGY}, volume={171}, ISSN={["0022-1899"]}, DOI={10.1093/infdis/171.5.1139}, abstractNote={The importance of venereal modes of B virus (cercopithecine herpesvirus 1) transmission was evaluated in 49 rhesus monkeys tested at necropsy. Antibodies to B virus were demonstrated in 19 monkeys, but no active viral shedding was detected in mucosal swabs collected at death. The polymerase chain reaction demonstrated presence of the ICP 18.5 (UL28) gene of B virus in neuronal tissues of 15 monkeys presumed to be latently infected, including 12.8% of trigeminal and 22.9% of lumbosacral ganglia pools. Two monkeys tested positive at both sites. Breeding history was predictive of B virus seropositivity (odds ratio, 1.64; 95% confidence interval, 1.21-2.23; P < .05). The population attributable risk of B virus seropositivity due to breeding was 22.7%, similar to the proportion of monkeys with B virus DNA in neuronal tissues subserving the genital region. Sexual contact is a significant, but not predominant, mode of B virus transmission between monkeys.}, number={5}, journal={JOURNAL OF INFECTIOUS DISEASES}, author={WEIGLER, BJ and SCINICARIELLO, F and HILLIARD, JK}, year={1995}, month={May}, pages={1139–1143} }
 @article{weigler_1995, title={Zoonotic hantaviruses: New concerns for the United States}, volume={206}, number={7}, journal={Journal of the American Veterinary Medical Association}, author={Weigler, B. J.}, year={1995}, pages={979} }