@article{hill_pullman_starcher_shih_1995, title={DIETARY NITROPRUSSIDE ALLEVIATES ATHEROSCLEROSIS IN HYPERCHOLESTEROLEMIC JAPANESE-QUAIL (COTURNIX-COTURNIX-JAPONICA)}, volume={112}, ISSN={["0300-9629"]}, DOI={10.1016/0300-9629(95)00076-J}, abstractNote={Male Japanese quail (Coturnix coturnix japonica), susceptible to cholesterol-induced atherosclerosis, were rendered hypercholesterolemic by feeding a diet containing 0.5% cholesterol in two experiments. Half the animals also received a dietary supplement of sodium nitroprusside ranging in concentration from 0.005% to 0.015%. After 10 weeks on the diets, serum was obtained for cholesterol analysis, the animals were killed, and the aortae removed and examined for the presence of atherosclerotic lesions. The number of animals having lesions and the severity of the lesions was reduced in a dose dependent manner among those animals receiving nitroprusside. Serum cholesterol was also reduced in response to increasing levels of dietary nitroprusside. These findings indicate that, in this model, dietary nitroprusside, a source of nitric oxide, can reduce the appearance and severity of atherosclerotic lesions in the aorta.}, number={1}, journal={COMPARATIVE BIOCHEMISTRY AND PHYSIOLOGY A-PHYSIOLOGY}, author={HILL, CH and PULLMAN, EP and STARCHER, B and SHIH, JCH}, year={1995}, month={Sep}, pages={151–154} }
 @article{hill_1994, title={INTERACTION OF VANADIUM AND PHOSPHORUS IN CHICKS}, volume={46}, ISSN={["0163-4984"]}, DOI={10.1007/BF02789302}, abstractNote={Studies were carried out to determine the effect of dietary vanadium on chicks fed phosphorus deficient and control diets. Vanadium at 50 mg/kg of diet decreased growth of both control and deficient chicks. The high mortality among the phosphorus deficient chicks was significantly alleviated by the presence of vanadium. The increased relative ventricular weights found among the deficient chicks was also alleviated by the presence of dietary vanadium. Vanadium fed at 10 or 20 mg/kg diet did not reduce growth rate but significantly reduced mortality among chicks fed the deficient diet and decreased the relative ventricle weights. Time course studies revealed that chicks are hatched with high relative ventricular weights (.83% of body wt) and remain at that level among chicks fed the phosphorus deficient diet. The addition of vanadium or phosphate to the diet resulted in a progressive decrease in relative ventricular weights. The inclusion of vanadium in the diet resulted in increased serum phosphorus levels among the deficient chicks that may be related to the decrease in mortality and relative ventricle weights.}, number={3}, journal={BIOLOGICAL TRACE ELEMENT RESEARCH}, author={HILL, CH}, year={1994}, month={Dec}, pages={269–278} }
 @article{hill_1992, title={Dietary vanadium and the oxidative state of hepatic and renal pyridine nucleotides}, volume={102}, DOI={10.1016/0300-9629(92)90025-L}, abstractNote={1. NAD, NADH, NADP and NADPH were measured in the livers and kidneys of chicks receiving 50 mg vanadium/kg diet. 2. There was no effect of dietary vanadium on the oxidative states of the nucleotides, although the growth rate was decreased. 3. The lack of effect of vanadium on the oxidative status of the nucleotides was ascribed to the low tissue concentration of vanadium.}, number={1}, journal={Comparative Biochemistry and Physiology. A, Physiology}, author={Hill, C. H.}, year={1992}, pages={139} }
 @article{hill_1990, title={Effect of dietary copper on vanadate toxicity in chicks}, volume={23}, DOI={10.1007/bf02917175}, abstractNote={The addition of copper to a corn-soybean diet at levels of 200 mg/kg and above lessened the growth-retarding effect of vanadate for chicks. This interaction between vanadate and copper was evident in both ad libitum-fed chicks and chicks in which feed consumption was restricted to approximately equal amounts. The ameliorating effect of copper was not accompanied by changes in the femur levels of vanadium nor by changes in the hepatic or renal glutathione concentrations. Zinc added at 515 mg/kg of diet had no effect on the toxicity of vanadium. Sodium sulfate added at a level to supply the same amount of sulfate, as supplied by 500 mg/kg copper sulfate, was without effect on the vanadate-induced growth depression. The underlying mechanism of the interaction of copper and vanadium is not known, but it does not lie in changes in feed consumption or organ burdens of vanadium, as represented by the femur vanadium concentrations.}, number={1}, journal={Biological Trace Element Research}, author={Hill, C. H.}, year={1990}, pages={17} }
 @article{hill_1990, title={Interaction of vanadate and chloride in chicks}, volume={23}, DOI={10.1007/bf02917173}, abstractNote={Six experiments have been conducted examining the interaction of dietary sodium chloride levels and vanadate toxicity in chicks. Increasing the dietary supplement of NaCl from 0.5 to 2.0% resulted in amelioration of vanadate toxicity, as measured by growth rate. The amelioration was found to reside in the chloride ion. Hepatic, renal, and femur vanadium concentrations were usually reduced at the higher levels of NaCl supplementation, but there was little correlation between these reductions and the reversal of vanadate toxicity.}, number={1}, journal={Biological Trace Element Research}, author={Hill, C. H.}, year={1990}, pages={1} }
 @article{hill_1990, title={The effect of dietary mercury on vanadate toxicity in the chick}, volume={23}, DOI={10.1007/bf02917174}, abstractNote={Three experiments were conducted investigating the interaction of dietary vanadate and mercury on the growth of chicks. The growth-retarding effect of 30 mg vanadium/kg diet was completely overcome by the inclusion of 500 mg mercury/kg diet. Restricting the feed intake of the mercury-supplemented animals to approximately those receiving vanadate alone still resulted in an amelioration of the growth retarding effect of vanadate. Analyses of femurs and kidneys revealed that mercury added to a vanadium-containing diet increased the vanadate concentration of the femur and had no effect on the vanadium concentration in the kidney. As little as 25 mg mercury/kg diet significantly reduced the growth retarding effect of vanadium. The inclusion of 100 mg mercury/kg in the diet resulted in a significant increase in renal glutathione concentration.}, number={1}, journal={Biological Trace Element Research}, author={Hill, C. H.}, year={1990}, pages={11} }
 @article{burt_hill_london_levine_1987, title={Magnetic resonance studies on the noncyclic phosphodiesters and other lipid related metabolites}, volume={1}, journal={FAEEB}, author={Burt, C. T. and Hill, C. H. and London, R. E. and Levine, J. F.}, year={1987}, pages={22–31} }