Hepatic β-oxidation and carnitine palmitoyltransferase I in neonatal pigs after dietary treatments of clofibric acid, isoproterenol, and medium-chain triglycerides
American Journal of Physiology-Regulatory, Integrative and Comparative Physiology, 288(6), R1518–R1524.
author keywords: acyl-CoA oxidase; fatty acid oxidase; malonyl-CoA; peroxisome; peroxisome proliferator; activated receptor
MeSH headings : Adrenergic beta-Agonists / pharmacology; Animals; Animals, Newborn / metabolism; Carnitine O-Palmitoyltransferase / metabolism; Clofibric Acid / pharmacology; DNA Primers; DNA, Complementary / biosynthesis; DNA, Complementary / genetics; Diet; Glucose / metabolism; Hypolipidemic Agents / pharmacology; Indicators and Reagents; Isoproterenol / pharmacology; Liver / metabolism; Mitochondria, Liver / enzymology; Mitochondria, Liver / metabolism; Oxidation-Reduction; Palmitic Acids / metabolism; Peroxisomes / metabolism; RNA, Messenger / biosynthesis; RNA, Messenger / genetics; Reverse Transcriptase Polymerase Chain Reaction; Swine; Triglycerides / pharmacology
topics (OpenAlex): Metabolism and Genetic Disorders; Peroxisome Proliferator-Activated Receptors; Adipose Tissue and Metabolism
TL;DR:
A suckling piglet model was used to study nutritional and pharmacologic means of stimulating hepatic fatty acid β-oxidation and its applications in nutrition and medicine.
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UN Sustainable Development Goal Categories
2. Zero Hunger
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