@article{jines_balint-kurti_robertson-hoyt_molnar_holland_goodman_2007, title={Mapping resistance to Southern rust in a tropical by temperate maize recombinant inbred topcross population}, volume={114}, ISSN={["1432-2242"]}, url={http://www.scopus.com/inward/record.url?eid=2-s2.0-33846813838&partnerID=MN8TOARS}, DOI={10.1007/s00122-006-0466-0}, abstractNote={Southern rust, caused by Puccinia polysora Underw, is a foliar disease that can severely reduce grain yield in maize (Zea mays L.). Major resistance genes exist, but their effectiveness can be limited in areas where P. polysora is multi-racial. General resistance could be achieved by combining quantitative and race-specific resistances. This would be desirable if the resistance alleles maintained resistance across environments while not increasing plant maturity. Recombinant inbred (RI) lines were derived from a cross between NC300, a temperate-adapted all-tropical line, and B104, an Iowa Stiff Stalk Synthetic line. The RI lines were topcrossed to the tester FR615 x FR697. The 143 topcrosses were scored for Southern rust in four environments. Time to flowering was measured in two environments. The RI lines were genotyped at 113 simple sequence repeat markers and quantitative trait loci (QTL) were mapped for both traits. The entry mean heritability estimate for Southern rust resistance was 0.93. A multiple interval mapping model, including four QTL, accounted for 88% of the variation among average disease ratings. A major QTL located on the short arm of chromosome 10, explained 83% of the phenotypic variation, with the NC300 allele carrying the resistance. Significant (P < 0.001), but relatively minor, topcross-by-environment interaction occurred for Southern rust, and resulted from the interaction of the major QTL with the environment. Maturity and Southern rust rating were slightly correlated, but QTL for the two traits did not co-localize. Resistance was simply inherited in this population and the major QTL is likely a dominant resistant gene that is independent of plant maturity.}, number={4}, journal={THEORETICAL AND APPLIED GENETICS}, author={Jines, M. P. and Balint-Kurti, P. and Robertson-Hoyt, L. A. and Molnar, T. and Holland, J. B. and Goodman, M. M.}, year={2007}, month={Feb}, pages={659–667} }
 @article{balint-kurti_krakowsky_jines_robertson_molnar_goodman_holland_2006, title={Identification of quantitative trait loci for resistance to southern leaf blight and days to anthesis in a maize recombinant inbred line population}, volume={96}, ISSN={["1943-7684"]}, url={http://www.scopus.com/inward/record.url?eid=2-s2.0-33749262148&partnerID=MN8TOARS}, DOI={10.1094/PHYTO-96-1067}, abstractNote={ A recombinant inbred line population derived from a cross between the maize lines NC300 (resistant) and B104 (susceptible) was evaluated for resistance to southern leaf blight (SLB) disease caused by Cochliobolus heterostrophus race O and for days to anthesis in four environments (Clayton, NC, and Tifton, GA, in both 2004 and 2005). Entry mean and average genetic correlations between disease ratings in different environments were high (0.78 to 0.89 and 0.9, respectively) and the overall entry mean heritability for SLB resistance was 0.89. When weighted mean disease ratings were fitted to a model using multiple interval mapping, seven potential quantitative trait loci (QTL) were identified, the two strongest being on chromosomes 3 (bin 3.04) and 9 (bin 9.03–9.04). These QTL explained a combined 80% of the phenotypic variation for SLB resistance. Some time-point-specific SLB resistance QTL were also identified. There was no significant correlation between disease resistance and days to anthesis. Six putative QTL for time to anthesis were identified, none of which coincided with any SLB resistance QTL. }, number={10}, journal={PHYTOPATHOLOGY}, author={Balint-Kurti, P. J. and Krakowsky, M. D. and Jines, M. P. and Robertson, L. A. and Molnar, T. L. and Goodman, M. M. and Holland, J. B.}, year={2006}, month={Oct}, pages={1067–1071} }
 @article{robertson-hoyt_jines_balint-kurti_kleinschmidt_white_payne_maragos_molnar_holland_2006, title={QTL mapping for fusarium ear rot and fumonisin contamination resistance in two maize populations}, volume={46}, DOI={10.2135/cropsci205.12-0450}, number={4}, journal={Crop Science}, author={Robertson-Hoyt, L. A. and Jines, M. P. and Balint-Kurti, Peter and Kleinschmidt, C. E. and White, D. G. and Payne, G. A. and Maragos, C. M. and Molnar, T. L. and Holland, J. B.}, year={2006}, pages={1734–1743} }
 @article{nelson_jines_goodman_2006, title={Selecting among available, elite tropical maize inbreds for use in long-term temperate breeding}, volume={51}, number={2}, journal={Maydica}, author={Nelson, P. T. and Jines, M. P. and Goodman, M. M.}, year={2006}, pages={255–262} }