@article{moskowitz_tracey_widmayer_dumont_2019, title={Meeting report: 32nd international mammalian genome conference Abstracts}, volume={30}, ISSN={["1432-1777"]}, DOI={10.1007/s00335-019-09797-1}, abstractNote={Over 150 scientists from more than 50 research institutions and eight countries attended the 32nd annual meeting of the International Mammalian Genome Society (IMGS) held in Rio Mar, Puerto Rico. Attendees included predoctoral and postdoctoral trainees, junior investigators, clinicians, industry professionals, and established leaders in mammalian genetics and genomics. From November 11–14, major scientific advances in the fields of systems genetics, developmental biology, cancer, human disease modeling, and bioinformatics were showcased in a series of 66 poster and 54 platform presentations. Here we provide an overview of the meeting’s proceedings and summarize the exciting, novel research findings communicated by conference participants that, collectively, are advancing the frontiers of mammalian genetics and genomics.}, number={3-4}, journal={MAMMALIAN GENOME}, author={Moskowitz, Jacob and Tracey, Lauren and Widmayer, Samuel and Dumont, Beth}, year={2019}, month={Apr}, pages={43–53} }
 @article{dumont_2017, title={Meiotic consequences of genetic divergence across the murine pseudoautosomal region}, volume={205}, number={3}, journal={Genetics}, author={Dumont, B. L.}, year={2017}, pages={1089–1100} }
 @article{dumont_2017, title={Variation and evolution of the meiotic requirement for crossing over in mammals}, volume={205}, number={1}, journal={Genetics}, author={Dumont, B. L.}, year={2017}, pages={155-} }
 @article{dumont_2017, title={X-chromosome control of genome-scale recombination rates in house mice}, volume={205}, number={4}, journal={Genetics}, author={Dumont, B. L.}, year={2017}, pages={1649–1656} }
 @article{roberts_juntti_coyle_dumont_stanley_ryan_fernald_roberts_2016, title={Polygenic sex determination in the cichlid fish Astatotilapia burtoni}, volume={17}, ISSN={["1471-2164"]}, DOI={10.1186/s12864-016-3177-1}, abstractNote={The East African riverine cichlid species Astatotilapia burtoni serves as an important laboratory model for sexually dimorphic physiology and behavior, and also serves as an outgroup species for the explosive adaptive radiations of cichlid species in Lake Malawi and Lake Victoria. An astounding diversity of genetic sex determination systems have been revealed within the adaptive radiation of East African cichlids thus far, including polygenic sex determination systems involving the epistatic interaction of multiple, independently segregating sex determination alleles. However, sex determination has remained unmapped in A. burtoni. Here we present mapping results supporting the presence of multiple, novel sex determination alleles, and thus the presence of polygenic sex determination in A. burtoni.Using mapping in small families in conjunction with restriction-site associated DNA sequencing strategies, we identify associations with sex at loci on linkage group 13 and linkage group 5-14. Inheritance patterns support an XY sex determination system on linkage group 5-14 (a chromosome fusion relative to other cichlids studied), and an XYW system on linkage group 13, and these associations are replicated in multiple families. Additionally, combining our genetic data with comparative genomic analysis identifies another fusion that is unassociated with sex, with linkage group 8-24 and linkage group 16-21 fused in A. burtoni relative to other East African cichlid species.We identify genetic signals supporting the presence of three previously unidentified sex determination alleles at two loci in the species A. burtoni, strongly supporting the presence of polygenic sex determination system in the species. These results provide a foundation for future mapping of multiple sex determination genes and their interactions. A better understanding of sex determination in A. burtoni provides important context for their use in behavioral studies, as well as studies of the evolution of genetic sex determination and sexual conflicts in East African cichlids.}, journal={BMC GENOMICS}, author={Roberts, Natalie B. and Juntti, Scott A. and Coyle, Kaitlin P. and Dumont, Bethany L. and Stanley, M. Kaitlyn and Ryan, Allyson Q. and Fernald, Russell D. and Roberts, Reade B.}, year={2016}, month={Oct} }
 @article{dumont_2015, title={Interlocus gene conversion explains at least 2.7 % of single nucleotide variants in human segmental duplications}, volume={16}, journal={BMC Genomics}, author={Dumont, B. L.}, year={2015} }
 @article{dumont_devlin_truempy_miller_singh_2015, title={No Evidence that Infection Alters Global Recombination Rate in House Mice}, volume={10}, ISSN={["1932-6203"]}, DOI={10.1371/journal.pone.0142266}, abstractNote={Recombination rate is a complex trait, with genetic and environmental factors shaping observed patterns of variation. Although recent studies have begun to unravel the genetic basis of recombination rate differences between organisms, less attention has focused on the environmental determinants of crossover rates. Here, we test the effect of one ubiquitous environmental pressure–bacterial infection–on global recombination frequency in mammals. We applied MLH1 mapping to assay global crossover rates in male mice infected with the pathogenic bacterium Borrelia burgdorferi, the causative agent of Lyme Disease, and uninfected control animals. Despite ample statistical power to identify biologically relevant differences between infected and uninfected animals, we find no evidence for a global recombination rate response to bacterial infection. Moreover, broad-scale patterns of crossover distribution, including the number of achiasmate bivalents, are not affected by infection status. Although pathogen exposure can plastically increase recombination in some species, our findings suggest that recombination rates in house mice may be resilient to at least some forms of infection stress. This negative result motivates future experiments with alternative house mouse pathogens to evaluate the generality of this conclusion.}, number={11}, journal={PLoS One}, author={Dumont, Beth L. and Devlin, Amy A. and Truempy, Dana M. and Miller, Jennifer C. and Singh, Nadia D.}, year={2015}, month={Nov} }
 @article{dumont_eichler_2013, title={Signals of historical interlocus gene conversion in human segmental duplications}, volume={8}, number={10}, journal={PLoS One}, author={Dumont, B. L. and Eichler, E. E.}, year={2013} }