Matthew Ian Gilmour

Works (5)

Updated: July 5th, 2023 15:54

2008 journal article

Modulation of pulmonary inflammatory responses and antimicrobial defenses in mice exposed to diesel exhaust

TOXICOLOGY AND APPLIED PHARMACOLOGY, 229(3), 310–319.

By: K. Gowdy n, Q. Krantz*, M. Daniels *, W. Linak*, I. Jaspers * & M. Gilmour *

author keywords: diesel exhaust; inflammation; lung; surfactant proteins; clara cell; mice
MeSH headings : Air Pollutants / toxicity; Animals; Cytokines / drug effects; Cytokines / metabolism; Female; Intercellular Adhesion Molecule-1 / drug effects; Intercellular Adhesion Molecule-1 / metabolism; Lung / drug effects; Lung / pathology; Mice; Mice, Inbred BALB C; Neutrophils / drug effects; Neutrophils / metabolism; Occupational Exposure / adverse effects; Pneumonia / chemically induced; Pulmonary Surfactant-Associated Protein A / drug effects; Pulmonary Surfactant-Associated Protein A / metabolism; Pulmonary Surfactant-Associated Protein D / drug effects; Pulmonary Surfactant-Associated Protein D / metabolism; Respiratory Tract Infections / etiology; Time Factors; Up-Regulation / drug effects; Uteroglobin / drug effects; Uteroglobin / metabolism; Vehicle Emissions / toxicity
TL;DR: Exposure to moderate and high occupational levels of DE caused an increase in lung injury and inflammation, and a decrease in host defense molecules, which could result in increased susceptibility to respiratory pathogens. (via Semantic Scholar)
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Source: Web Of Science
Added: August 6, 2018

2007 journal article

Diesel exhaust enhanced susceptibility to influenza infection is associated with decreased surfactant protein expression

INHALATION TOXICOLOGY, 19(14), 1121–1133.

By: J. Ciencewicki*, K. Gowdy n, Q. Krantz*, W. Linak *, L. Brighton*, M. Gilmour  n, I. Jaspers

MeSH headings : Animals; Female; Gene Expression Regulation / physiology; Genetic Predisposition to Disease; Humans; Influenza, Human / etiology; Influenza, Human / genetics; Influenza, Human / metabolism; Mice; Mice, Inbred BALB C; Pulmonary Surfactant-Associated Proteins / biosynthesis; Pulmonary Surfactant-Associated Proteins / genetics; Pulmonary Surfactant-Associated Proteins / physiology; Pulmonary Surfactants / metabolism; Vehicle Emissions / toxicity
TL;DR: Exposure to DE enhances the susceptibility to respiratory viral infections by reducing the expression and production of antimicrobial surfactant proteins, which are components of the interferon-independent antiviral defenses. (via Semantic Scholar)
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Source: Web Of Science
Added: August 6, 2018

2003 article

Role of oxidative stress and p38MAP kinase in murine pulmonary responses to LPS and diesel exhaust particles.

American Journal of Respiratory and Critical Care Medicine, Vol. 167, p. A622.

By: P. Singh, C. Dick, E. Boykin, D. Andrews, K. Adler & I. Gilmour

Source: NC State University Libraries
Added: August 6, 2018

2002 article

Diesel exhaust particles increase lung injury and inflammation during early sensitization in brown Norway rats.

Toxicological Sciences, Vol. 66, p. 147.

By: P. Singh, M. Daniels, D. Winsett, J. Richards, L. Bishop, C. Dick, M. Madden, K. Adler, M. Gilmour

Source: NC State University Libraries
Added: August 6, 2018

2002 article

Effects of allergic airways disease on influenza virus infection in brown Norway rats.

American Journal of Respiratory and Critical Care Medicine, Vol. 165, p. A183.

By: P. Singh, D. Winsett, M. Daniels, C. Dick, K. Adler & M. Gilmour

Source: NC State University Libraries
Added: August 6, 2018

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