Jo-David Fine

Works (3)

Updated: July 5th, 2023 16:03

2001 journal article

Enhanced expression of mucin genes in a guinea pig model of allergic asthma

AMERICAN JOURNAL OF RESPIRATORY CELL AND MOLECULAR BIOLOGY, 25(5), 644–651.

By: Y. Li n, L. Martin n, M. Minnicozzi n, S. Greenfeder n, J. Fine n, C. Pettersen n, B. Chorley  n, K. Adler n

MeSH headings : Amino Acid Sequence; Animals; Antineoplastic Agents / pharmacology; Asthma / physiopathology; Base Sequence; Bronchial Hyperreactivity / physiopathology; Cells, Cultured; Chemokine CCL11; Chemokines, CC / genetics; Cloning, Molecular; Conserved Sequence; Disease Models, Animal; Eosinophils / immunology; Gene Expression / physiology; Guinea Pigs; Histamine / pharmacology; Hypersensitivity / physiopathology; Inflammation Mediators / pharmacology; Molecular Sequence Data; Mucin 5AC; Mucin-2; Mucins / genetics; Ovalbumin / immunology; Ovalbumin / pharmacology; RNA, Messenger / analysis; Respiratory Mucosa / cytology; Respiratory Mucosa / physiology; Trachea / cytology; Tumor Necrosis Factor-alpha / pharmacology
TL;DR: Results of these studies suggest that the OVA-sensitized guinea pig responds to allergic challenge with enhanced expression of genes that likely play a role in increased airway inflammation and mucin overproduction, and enhanced mucin gene expression appears to occur before eosinophil infiltration. (via Semantic Scholar)
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Added: August 6, 2018

1998 journal article

Canine epidermolysis bullosa acquisita: circulating autoantibodies target the aminoterminal noncollagenous (NC1) domain of collagen VII in anchoring fibrils

VETERINARY DERMATOLOGY, 9(1), 19–31.

By: T. Olivry n , J. Fine*, S. Dunston n, D. Chasse n, A. Tenorio*, N. Monteiro-Riviere n , M. Chen *, D. Woodley*

Contributors: T. Olivry n , J. Fine*, S. Dunston n, D. Chasse n, A. Tenorio*, N. Monteiro-Riviere n , M. Chen *, D. Woodley*

author keywords: epidermolysis bullosa acquisita; dog; canine; collagen VII; anchoring fibril; blistering disease; autoimmunity
TL;DR: This study establishes the existence of the canine equivalent of epidermolysis bullosa acquisita (EBA) in humans, and confirms that the circulating autoantibodies are specific for the aminoterminal globular non-collagenous NC1 domain of type VII collagen. (via Semantic Scholar)
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Sources: Web Of Science, ORCID, NC State University Libraries
Added: August 6, 2018

1998 journal article

Uncein may be a potential target for sulfur mustard alkylation

TOXICOLOGY METHODS, 8(1), 27–36.

By: Z. Zhang n, J. Fine* & N. Monteiro-Riviere n 

Contributors: Z. Zhang n, J. Fine* & N. Monteiro-Riviere n 

author keywords: basement membrane; blister; cell culture; epidermal-dermal junction; keratinocytes; sulfur mustard; uncein; vesication
TL;DR: Uncein is a newly discovered anchoring filament-associated antigen that has been shown to be undetectable in all forms of junctional epidermolysis bullosa, therefore suggesting its role in maintaining the integrity of the epidermal-dermal basement membrane zone. (via Semantic Scholar)
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UN Sustainable Development Goal Categories
Sources: Web Of Science, ORCID, NC State University Libraries
Added: August 6, 2018

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