Jo-David Fine

Works (3)

Updated: July 5th, 2023 16:03

2001 article

Enhanced Expression of Mucin Genes in a Guinea Pig Model of Allergic Asthma

Li, Y., Martin, L. D., Minnicozzi, M., Greenfeder, S., Fine, J., Pettersen, C. A., … Adler, K. B. (2001, November 1). American Journal of Respiratory Cell and Molecular Biology.

By: Y. Li n, L. Martin n, M. Minnicozzi n, S. Greenfeder n, J. Fine n, C. Pettersen n, B. Chorley  n, K. Adler n

MeSH headings : Amino Acid Sequence; Animals; Antineoplastic Agents / pharmacology; Asthma / physiopathology; Base Sequence; Bronchial Hyperreactivity / physiopathology; Cells, Cultured; Chemokine CCL11; Chemokines, CC / genetics; Cloning, Molecular; Conserved Sequence; Disease Models, Animal; Eosinophils / immunology; Gene Expression / physiology; Guinea Pigs; Histamine / pharmacology; Hypersensitivity / physiopathology; Inflammation Mediators / pharmacology; Molecular Sequence Data; Mucin 5AC; Mucin-2; Mucins / genetics; Ovalbumin / immunology; Ovalbumin / pharmacology; RNA, Messenger / analysis; Respiratory Mucosa / cytology; Respiratory Mucosa / physiology; Trachea / cytology; Tumor Necrosis Factor-alpha / pharmacology
topics (OpenAlex): Asthma and respiratory diseases; Antimicrobial Peptides and Activities; Biochemical Analysis and Sensing Techniques
TL;DR: Results of these studies suggest that the OVA-sensitized guinea pig responds to allergic challenge with enhanced expression of genes that likely play a role in increased airway inflammation and mucin overproduction, and enhanced mucin gene expression appears to occur before eosinophil infiltration. (via Semantic Scholar)
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Source: Web Of Science
Added: August 6, 2018

1998 article

Canine epidermolysis bullosa acquisita: circulating autoantibodies target the aminoterminal non‐collagenous (NC1) domain of collagen VII in anchoring fibrils

OLIVRY, T. H. I. E. R. R. Y., FINE, J. O. D. A. V. I. D., DUNSTON, S. T. A. N. L. E. Y. M., CHASSE, D. A. W. N., TENORIO, A. U. R. E. A. P. A. S. C. A. L., MONTEIRO‐RIVIERE, N. A. N. C. Y. A., … WOODLEY, D. A. V. I. D. T. (1998, March 1). Veterinary Dermatology, Vol. 9, pp. 19–31.

By: T. Olivry n , J. Fine*, S. Dunston n, D. Chasse n, A. Tenorio*, N. Monteiro‐Riviere n , M. Chen *, D. Woodley*

Contributors: T. Olivry n , J. Fine*, S. Dunston n, D. Chasse n, A. Tenorio*, N. Monteiro-Riviere n , M. Chen *, D. Woodley*

author keywords: epidermolysis bullosa acquisita; dog; canine; collagen VII; anchoring fibril; blistering disease; autoimmunity
topics (OpenAlex): Autoimmune Bullous Skin Diseases; Skin and Cellular Biology Research; Coagulation, Bradykinin, Polyphosphates, and Angioedema
TL;DR: This study establishes the existence of the canine equivalent of epidermolysis bullosa acquisita (EBA) in humans, and confirms that the circulating autoantibodies are specific for the aminoterminal globular non-collagenous NC1 domain of type VII collagen. (via Semantic Scholar)
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Sources: Web Of Science, ORCID, NC State University Libraries
Added: August 6, 2018

1998 article

UNCEIN MAY BE A POTENTIAL TARGET FOR SULFUR MUSTARD ALKYLATION

Zhang, Z., Fine, J.-D., & Monteiro-Riviere, N. A. (1998, January 1). Toxicology Methods, Vol. 8, pp. 27–36.

By: Z. Zhang n, J. Fine* & N. Monteiro-Riviere n 

Contributors: Z. Zhang n, J. Fine* & N. Monteiro-Riviere n 

author keywords: basement membrane; blister; cell culture; epidermal-dermal junction; keratinocytes; sulfur mustard; uncein; vesication
topics (OpenAlex): Skin and Cellular Biology Research; Contact Dermatitis and Allergies; Mast cells and histamine
TL;DR: Uncein is a newly discovered anchoring filament-associated antigen that has been shown to be undetectable in all forms of junctional epidermolysis bullosa, therefore suggesting its role in maintaining the integrity of the epidermal-dermal basement membrane zone. (via Semantic Scholar)
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Sources: Web Of Science, ORCID, NC State University Libraries
Added: August 6, 2018

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